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Furthermore, even in p53-mutant conditions, the cell cycle can be blocked as p27 levels increase through p53-independent mechanisms, including reduced AKT-mediated degradation and FoxO transcription factor activation

doi: 10.1097/01.jcp.0000095340.32154.c6

Prevalence and zoonotic risks of Trichophyton mentagrophytes and Cheyletiella spp

10.1002/anie.202111853 Angew Chem

LongYQiuJZhangBHePShiXHeQet alPharmacological vitamin C treatment impedes the growth of endogenous glutamine-dependent cancers by targeting glutamine synthetase