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Jean Marcoux, MD, FRCP(C)

HFD can metabolic dysfunction-associated steatotic liver disease (MASLD) by increasing the uptake of free fatty acids by hepatocytes, disrupting the balance between de novo synthesis and oxidative degradation of fatty acids, leading to excessive deposition of triglycerides (TG) in hepatocytes

409,410,411 In addition, molecular mimicry between EBV proteins and autoantigens provides another possible model for disease induction and immune escape

doi: 10.1186/s13195-019-0535-7 141 MengelD.DamsJ.ZiemekJ.BeckerJ.Balzer-GeldsetzerM.HilkerR.et al

Compr Physiol 5(2):649665

Indeed, we performed RNA-seq of 5 hyperplastic and 3 normal prostate specimens and found that there was a number of DEGs between hyperplastic prostate and normal prostate, of which the up-expressed matrix-remodeling associated 5 (MXRA5) [9], C-X-C motif chemokine ligand 13 (CXCL13) [10], bone morphogenetic protein 5 (BMP5) [11] and neural epidermal growth factor-like like 2 (NELL2) [12], and the down-expressed glutathione peroxidase 3 (GPX3) ranked at the top and most of them have been reported