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Complex changes of GABAA and GABAB receptor binding in the spinal cord dorsal horn following peripheral inflammation or neurectomy

Paradoxically, this is accompanied by a BDNF-TrkB-mediated upregulation of synaptic GABA A Rs and by an 1-to-2GABA A R subunit switch, providing a mechanistic rationale for the analgesic action of the 2,3GABA A R benzodiazepine-site ligand L838,417 after nerve injury

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The self-assembled nature of these nanoparticles not only addresses the instability issues of GHK-Cu in biological fluids but also enhances their antibacterial efficacy

Crystal structure of the human 2 adrenergic G-protein-coupled receptor