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10.1007/s11356-019-07511-x 160 VizsnyiczaiG.FrangipaneG.BianchiS.SaglimbeniF.DellArcipreteD.Di LeonardoR

Failures in drug development can often be attributed to gaps in our understanding of the biological underpinnings of human diseases, an overreliance on nonhuman models, and deficiencies such as low specificity, suboptimal pharmacological properties, or severe adverse reactions to drugs

Additional drugs such as the nerve protective agent olesoxime and the ion channel-targeting neurosteroids have been established promising effect on CIPN in preclinical studies, which, however, are required to be further confirmed in human studies (Table 1 )

Large alpha-synuclein oligomers inhibit neuronal SNARE-mediated vesicle docking

Having previously conducted a series of investigations of the oxidative damage theory that did not support it [34-38], we were curious about the life-extending effect of GSH, which is consistent with this theory