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Description

The data suggest that DUSP3 downregulation may contribute to macrophage-mediated inflammation of RA patients

The control group showed no significant changes

The mechanisms underlying ferroptosissuch as iron overload, lipid metabolism dysregulation, and antioxidant defense failureare increasingly recognized as key contributors to neuronal loss and functional deterioration in these disorders

Mechanistically, the abnormal proliferation of these tumor cells was due to the reduction of Asp synthesis through inhibiting Gln oxidation and attenuating reductive carboxylation following GOT1 deletion, which disrupted nucleotide generation within the cells