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Third, translational gaps between preclinical models and clinical practice are prominent: most therapeutic candidates (e.g., natural products and repurposed drugs) have only been validated in rodent models, with limited data on human renal tissue responses, optimal dosing windows, or potential drugdrug interactions in clinical settings (e.g., in patients undergoing kidney transplantation or cardiovascular surgery)

(Figure 3B), which is consistent with our previous results

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However, advanced peptide testing can detect it in more specific studies or research scenarios

Chromatin breakdown by deoxyribonuclease1 promotes acetaminophen-induced liver necrosis: an ultrastructural and histochemical study on male CD-1 mice

Fourth, the translation of mechanistic insights from animal or in vitro models remains tentative, as these systems may not recapitulate the complex interplay of metabolic, hormonal, and immune factors in human PCOS