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Increased G6PD activity, which is the gatekeeping enzyme of the pentose phosphate pathway [69], and xanthine suggest an increased turnover rate for purine nucleosides, which may arise from the increased DNA damage from continuous exposure to tamoxifen

Clin Nutr 35:175182

In eukaryotes, protein S -glutathionylation has emerged as major redox-regulatory mechanism that controls the activity of redox sensing transcription factors and protects active site Cys residues against irreversible oxidation to sulfonic acids (Dalle-Donne et al., 2009)

10.1104/pp.113.223545 68 NutricatiE.MiceliA.BlandoF.De BellisL

Lowering GSH levels also triggers ferroptosis through deactivation of GPX4, leading to accumulation of lipid peroxides [87] In some cases, GSH depletion alone can induce therapeutic effects without relying on Fenton chemistry, which typically involves HO reacting with Fe 2 to form hydroxyl radicals (OH) that damage critical biomolecules such as DNA, proteins, and lipids

& Araki, S