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Protein misfolding and failure of clearance systems, including the autophagy-lysosome and ubiquitin-proteasome pathways, further compromise the cellular environment and lead to toxic intracellular accumulations [31, 32]

The change from uncompetitive to non-competitive mechanism induced by chemical modifications of the inhibitors is not rare in drug design studies 48,49,50

Int Immunopharmacol 73:539551

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The increase of intracellular ROS is believed to correlate with the activation of nuclear factor (NF)-kappa B, a transcription activator linked to the elaboration of several cytokines (Figure 6)

ATRX deficiency has been shown to accelerate GBM growth and reduce survival, linking ATRX loss to DNA repair deficiencies, particularly impaired nonhomologous end joining