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This is supported by some of the existing research on the compound

10.1007/s00586-013-2767-8 264 SivanS

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View Abstract Strain induced esophageal growth in a novel rodent model

When lysosomal function is severely impaired, lysosome-independent degradation pathways, such as those activated by Bortezomib, a proteasome modulator, can enhance ubiquitination and proteasomal activity, compensating for defective lysosomal function and facilitating the removal of damaged proteins and organelles, thus improving immunotherapy efficacy (133) ( Table 1 )