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Endothelial dysfunction, including decreased endothelial cell-dependent vasorelaxation, is increasingly proposed to be triggered by free radicals generated during auto-oxidation of homocysteine (Loscalzo, 1996)

This peptide also appeared to modulate immune cell behavior, with reports of increased macrophage activity, which may support resolution rather than persistence of inflammation

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Sauer SW, Opp S, Hoffmann GF, Koeller DM, Okun JG, Klker S

It has been studied extensively in laboratory and animal models for its ability to accelerate healing, particularly within the digestive system and connective tissues