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Conclusions Nrf2 is a master regulator of several protective stress response pathways and it is not surprising that it became a highly competitive target for pharmacological modulation (Cuadrado et al

Serum samples were diluted twofold in sample dilution buffer

Rather than targeting one receptor or one pathway (like most drugs), GHK-Cu orchestrates a coordinated shift in gene expression across 31.2% of the genome

Am J Dis Child 141:10871089

GHK-CU boosts endothelial cell tube formation in vitro and increases blood vessel density in wound healing models