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Future therapeutic strategies should pivot from mere receptor blockade to targeting the core upstream drivers of checkpoint expression, such as epigenetic reprogramming, key cytokine signaling, or metabolic dysregulation, to fundamentally reverse the T-cell exhaustion program

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This dysfunction cascade further compromises cellular integrity and energy-dependent physiological functions such as ion channel activity and synaptic transmission [54, 55]

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It is not approved for human or animal use and must not be incorporated into foods, cosmetics, medications, or diagnostic procedures