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Disease-associated microglia: a universal immune sensor of neurodegeneration

Growth retardation was observed in rats after two generations of selenium deficiency 62

We speculate that this specific residue, which is located in the linker region of human parkins sequence, could play a key role in its folding, in interactions with other molecules, and thus in its functions, akin to the phosphorylation at serine 65 during toxin-induced mitophagy in mammalian cells [3, 30, 34]