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Future therapeutic strategies should pivot from mere receptor blockade to targeting the core upstream drivers of checkpoint expression, such as epigenetic reprogramming, key cytokine signaling, or metabolic dysregulation, to fundamentally reverse the T-cell exhaustion program

Yao ZW, Zhu H

Pervasive axonal transport deficits in multiple sclerosis models

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Our aim was to compare proteins involved in cell death from different defense pathways by TMV and its faster replicating mutant TMV 24A + UPD

Targeting multiple stages of the viral life cycle (such as entry, replication, and assembly) through multitarget drug design can help mitigate the risk of resistance