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It plays a crucial role in energy production by transporting fatty acids into mitochondria, where they are burned to generate ATP, the body's primary energy source

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There is evidence that oxidative stress may reduce corticosteroid sensitivity in COPD by mechanisms involving phosphorylation of the glucocorticoid receptor (GR) as well as phosphorylation and inactivation of histone deacetylase-2 (HDAC-2) [38,39,40,41]