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This work was supported by the Wilmot Cancer Institute Predoctoral Fellowship (to G.A.), the American Association for Cancer Research and Breast Cancer Research Foundation (20-20-26-HARR to I.S.H.), the Breast Cancer Coalition of Rochester (to I.S.H.), the American Cancer Society (RSG-23-971782-01-TBE to I.S.H.), the Ludwig Center for Metastasis Research (to A.M.), the University of Chicago Comprehensive Cancer Center (to A.M.), the Cancer Research Foundation (to A.M.), the V Foundation (to A.M.), the Pancreatic Cancer Action Network (to G.C.), the Lung Cancer Research Foundation (to J.C.), and NIH grants R01CA269813 (to I.S.H.), R01CA269813-S1 (to M.Z.), R37CA230042 (to G.M.D.), R24AA022057 (to V.V.), R01AA028859 (to Y.C.), R01CA276461 (to A.M.), T32CA009594 and F31CA278362 (to C.S.), and AI150698 (to J.M.)

Earlier studies have demonstrated that UT6 is responsible for binding with Npl4 and Cdc48/p97 24,41

Ketones block amyloid entry and improve cognition in an Alzheimers model

This progress was greatly facilitated by the identification of a specific motif recognized by AMPK

REV-ERB controls autophagy which is modulated in a circadian manner