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The administration of ROS scavengers, such as N-acetylcysteine, significantly mitigated the effect of alcohol-induced ROS and histone H3 acetylation, whereas the administration of ROS inducers and inhibitors of mitochondrial complexes I and III (rotenone and antimycin, respectively) enhanced histone H3 acetylation at Lys9

Abstract The future GCC-connected environmental risk factors expedited the progression of nCDs

Endometriosis and autoimmunity

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The transcriptomic divergence could simply reflect the difference between acute chemical shock and steady-state adaptation