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In fact, as discussed recently, in the absence of estrogen receptor, the FSP1-CoQ10-NAD(P)H axis was proposed to be a key process ensuring the maintenance of this homeostasis, enabling cell survival [26]

We show that senescent cells are primed for ferroptosis, displaying high levels of oxidative stress and intracellular Fe 2+ , but also upregulate GPX4, which prevents the accumulation of oxidized lipids

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Previous studies showed that VMP1 overexpression alleviates choline-deficient amino acid-defined high-fat diet (CDAHFD)-induced NASH in mice (PMID:35452693)