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Interestingly, increased GSTP1 expression has been shown to maintain the CSC phenotype in lung adenocarcinoma and facilitated metastasis through regulating EMT (Wang et al

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This IRAK/TRAF6 complex dissociates from TLR4, interacts with TGF--activating kinase-1 (TAK1) and its associated TAK-binding proteins, and thus forms the TRAF6/TAB/TAK1 complex (80)