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This rodent model can be divided into two phases: (1) initial demyelination in the corpus callosum and other brain regions, such as the superior cerebellar peduncles and cerebral cortex (Vega-Riquer, 2019), which is accompanied by activation of the innate immune system, and (2) significant remyelination of exposed axons once cuprizone treatment is terminated (Gharagozloo, 2022)

3a, we observed blunted expression of both gst-4::GFP and glod-4::GFP when animals consumed the glucose-fed bacteria, as well as an increase in sod-3::GFP