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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

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How It Works GHKCu is a naturally occurring copperbinding tripeptide that penetrates the stratum corneum and signals fibroblasts to produce more collagen, elastin, and glycosaminoglycans while regulating matrix metalloproteinases to reduce excessive collagen breakdown

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More substantial cognitive benefits, including memory enhancement and improved learning capacity, typically develop over 6 to 12 weeks as synaptic density increases