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Ferroptosis inhibitors act by inhibiting iron accumulation and reducing iron overload (e.g., deferoxamine [DFO]) or by inhibiting lipid peroxidation and reducing lipid ROS production (e.g., liproxstatin-1, vitamin E, and ferrostatin1 [Fer-1]) ( DFO has been shown to ameliorate cardiomyocyte injury in an in vitro DCM model of ECT exposed to AGE, reducing the expression of the lipid peroxidation marker malondialdehyde (MDA) and the ferroptosis marker prostaglandin-endoperoxide synthase-2 (PTGS 2 ), improving ECT cardiomyocyte function

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Li MS, Adesina SE, Ellis CL, Gooch JL, Hoover RS, Williams CR

doi: 10.1016/0003-3472(77)90048-3 32 FernaldR

This type of increase is expected and does not usually require intervention