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Description

Neurology 1981;25(1):16-24

10.3389/fncel.2022.836931 207 WatanabeT.NagaseK.ChosaM.TobinaiK

doi:10.1136/jclinpath-2021-20789539 40

In a CS-exposed COPD mouse model, SIRT1 deficiency led to increased acetylation levels of Forkhead Box O3(FOXO3), decreased PINK1 protein levels, and weakened mitophagy, which further exacerbated cellular senescence and airway resistance

found that sunitinib produced cardiac toxicity via the involvement of fibrotic and inflammation processes mediated by transcription factor NF-kB, and they also reported that l-carnitine showed a protective effect against secondary fibrotic process, cardiac inflammation, and arterial hypertension induced with sunitinib in Wistar rats [83]