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These findings pertain to the internal mechanisms underlying neuroprotection and reduction of oxidative stress

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Therapeutic strategies that enhance antioxidant capacity (e.g., hyperbaric oxygen[74]), modulate metabolism and inflammation (e.g., adenosine[75]), or reduce cardiac workload (e.g., atenolol[72]) have shown protective potential, highlighting the importance of targeting metabolic instability and oxidative damage

Despite the above-mentioned discrepancy, it appears that GHRs are central to the regulation of the quinone redox state, likely preventing toxicity of quinones, either naturally present or found as environmental pollutants

The oncometabolite R-2-hydroxyglutarate activates NF-B-dependent tumor-promoting stromal niche for acute myeloid leukemia cells

Reduced synaptic transmission and intrinsic excitability of a subtype of pyramidal neurons in the medial prefrontal cortex in a mouse model of Alzheimers disease