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We also showed that the secretome of differentiated adipocytes coming from tumor-bearing women was more enriched in PA and decreased in OL compared to the secretome coming from tumor-free breast adipose tissue [13]

[58] discovered that a subset of patients with ME/CFS displayed B cell hyperactivity, indicating its potential contribution to disease pathogenesis via autoimmune-mediated mechanisms

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The emergence of this detrimental cycle relies on dysregulation within the gut-organ axes