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3.9 Summary Taken together, the available experimental evidence supports a hierarchical model of AP pathogenesis centered on the mitochondrial hub ( Figure 3 ), in which early pathogenic triggers, including toxic calcium signals, bile acid exposure, alcohol metabolites, hyperstimulation, and lipotoxic stress, initiate progressive remodeling of mitochondrial contact sites with the ER, lysosomes, the cytoskeleton, and other organelles

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It enhances their chemosensitivity to conventional therapies [230, 231]

All data were generated in-house, and no paper mill was used

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