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When Mito-Exosomes are engulfed by alveolar macrophages, the mtDNA loaded within acts as a potent Damage-Associated Molecular Pattern (DAMP), escaping lysosomal degradation and activating the cyclic GMP-AMP synthase-stimulator of interferon genes (cGAS-STING) pathway or TLR9 signaling axis (51, 52)

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This means that ferroptosis can induce other kinds of cell death, and/or it plays a minor effect in cell death, or we use inhibitors after the reversible point and once this threshold is exceeded, ferroptosis will be irreversible Recently, we also found that ferroptosis is vital for tumor immunity such as macrophage phagocytosis (71) and T cell killing (115) but the crosstalk between ferroptosis and the glioma immunosuppressive microenvironment is not clear