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For research framed around overlapping metabolic-aging pharmacology, see also the related compound hubs for MOTS-c (mitochondrial-derived peptide regulating AMPK and metabolic-aging endpoints) and NAD+ (NAD + salvage substrate directly elevating cellular NAD + pools)

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In conclusion, our data suggest that the treatment with the antioxidant peptide Epitalon enhanced impaired wound healing processes in HG-injured ARPE-19 cells by inhibiting hyperglycemia-induced EMT and fibrosis, thus supporting the therapeutic benefits of this interesting peptide for DR management