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For parthanatos, DNA damage caused by mitochondrial impairment activates poly(ADP-ribose) polymerase 1 (PARP1), and excessive production of poly(ADP-ribose) (PAR) leads to NAD+ depletion and mitochondrial function collapse, ultimately inducing parthanatos [42]

10.1016/j.bbagen.2013.04.023 118 SteibK.SchffnerI.JagasiaR.EbertB.LieD

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In preclinical research models, exposure to GHK-Cu has been associated with altered gene expression patterns and modulation of signalling pathways related to extracellular matrix dynamics, oxidative stress response, and cellular turnover

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