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Many studies have confirmed a crucial role of N-type and T-type Ca v channels in paclitaxel-induced CIPN [74] as well as a key role of T-type Ca v channels in CIPN caused by vincristine [89]

We observed that H 2 O 2 treatment caused a significant decrease in total glutathione compared to non-treated animals, but this parameter was partially restored in the presence of GA (51% [ p = 0.0457] and 91% [ p = 0.1231] of the control, respectively)

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