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Although the above mechanisms have been discovered and confirmed repeatedly, therapeutic strategies should be more specifically targeted to mitochondrial energy defects rather than the neuroprotective effects of antioxidants [52]

[DOI] [PMC free article] [PubMed] [Google Scholar] 28.Hansen M, Rubinsztein DC & Walker DW Autophagy as a promoter of longevity insights from model organisms

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Based on the results of a previous study, to reduce unexpected severe toxicities, we comprehensively evaluated Hcy, B12, and FA levels and other clinical features, such as non-hematological or nutritional markers and genetic features

AOD-9604 is well soluble, any turbidity indicates degradation or contamination

BMC Neurol 11:139