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fumigatus has demonstrated an ability to bind Factor H, FHL-1, and C4BP on their surface to down-regulate the complement cascade,27,180,288 and to produce a soluble complement-inhibitory factor, which may be lipid derived, that prevented the activation of the alternative pathways.293,294 Moreover the thick fungal cell wall is largely resistant to direct lysis by the terminal membrane attack complex of the complement system.132 Various different types of behaviour have been detected on activation of immune cells through Toll-like receptors (TLR) by conidia and hyphae of this fungus

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