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This differs from direct GH replacement, where external hormone administration can affect natural production pathways

So, the modified Mod GRF 1-29 with increased binding affinity to GHRH receptors and prolonged half-life was developed, too

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These antiapoptotic mechanisms constitute vulnerabilities and, consequently, targeting them with either BCL2 family inhibitors 24,25,34 or via suppression of cFLIP 26 kills senescent cells