Adam Zahand 1 , Byron Williams 1 , and Jun Liu 1 1 Department of Molecular Biology and Genetics, Cornell University, Ithaca, NY, USA Correspondence to: Jun Liu ([email protected]) Abstract Description Methods Acknowledgements Funding Author Contributions Adam Zahand: Writing - review & editing, Writing - original draft, Visualization, Investigation Byron Williams: Investigation, Writing - review & editing Jun Liu: Conceptualization, Investigation, Project administration, Funding acquisition, Writing - review & editing, Supervision, Writing - original draft Nomenclature Validated By Daniela Raciti Database Reference ID: WBPaper00067132 History Received: 7/24/2024 Revision Received: 8/11/2024 Accepted: 8/10/2024 Published Online: 8/15/2024 Indexed: 8/29/2024 References Copyright 2024 by the authors
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Although no significant lipid peroxidation is observed in cancer-associated muscle atrophy, increased mitochondrial protein carbonylation impairs mitochondrial function, suggesting that regulating mitochondrial phospholipid biosynthesis pathways may be an effective therapeutic target for cancer-associated muscle wasting (81)
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