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Long-term (24 weeks) therapy in BALbc.Mdr2-/- model resulted in moderate 25% reduction in bridging fibrosis, but not in net collagen deposition

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A key pathological characteristic of this condition is the three-layer remodeling of distal pulmonary vessels, which involves excessive growth of endothelial cells, smooth muscle cells, and fibroblasts, along with the presence of inflammatory cell infiltration [27,28,29]

[2] However, later studies failed to demonstrate a sufficient lipolytic effect