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Therefore, a rise in Tfr cell levels in both peripheral blood and BALF in sarcoidosis may be a compensatory response necessary to curb inflammation at the site of the tissue lesion, where Tfr cell functional activity may not be as effective, which could contribute to pulmonary fibrosis

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DNA-based nanostructured platforms as drug delivery systems

Furthermore, we highlight the therapeutic potential of targeting these specific cell death mechanisms, which may facilitate the development of uveitis-specific biomarkers and inform future strategies for targeted combination therapies (see Table 2 )