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Some cytosolic GSH is transported, thanks to a specic transporter, the multidrug resistance protein 1 (MRP1), out of the cell to maintain a balance in the redox level [ 22 ], or can be conjugated by GSH transferases (GST) with endogenous metabolites, such as cysteinyl leukotriene or xenobiotics (therapeutics, anticancer drugs, carcinogens, pesti- cides, herbicides), forming a complex GSH-Xen, which is transported outside the cell by MRP1 [19]

A comprehensive diagnostic evaluation includes a detailed medical history, physical examination, and screening for antiphospholipid syndrome and uterine anomalies ( A variety of strategies are integrated within the therapeutic frameworks addressing RM

The two compounds therefore have no shared metabolic pathway that could create a clinically meaningful kinetic interaction

35 Because a number of bacterial proteins are Mn dependent, host-mediated Mn sequestration has the potential to disrupt bacterial pathogenesis

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Wen L, Shi D, Zhou T, Tu J, He M, Jiang Y, Yang B (2020) Identification of two novel prenylated flavonoids in mulberry leaf and their bioactivities