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This effect usually occurs immediately

The activation of HO-1, a mitochondrial enzyme that catalyses the degradation of haem to generate ferrous iron, results in increased mitochondrial iron, thereby upregulating ferroptosis [139, 140]

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Significantly differential pathways also point to enhanced methionine consumption and SAH elevation in supercooled E-Sol5 storage

This helps researchers assess the compound's effects more clearly and observe any lasting changes after administration ceases