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Sci Rep 12:103

Excessive lipid reprogramming increases the proportion of PUFAs within phospholipids, providing the fuel for ferroptosis

Second, the upstream dominance of ferroptosis within the PD cell death network is not fully validated

increase, decrease, tumor inflammatory genes Gremlin (GREM1), interleukin 1 (IL-1), interleukin-4 [IL-4], nuclear factor B subunit 1 (NFB1), and prostaglandin-endoperoxide synthase 2 (PTGS2), extracellular signal-regulated kinase (ERK), c-jun N-terminal kinase (JNK), light chain 3 protein B II (LC3BII), c-Jun N-terminal kinase (JNK), poly(ADP ribose) polymerase (PARP)-1, nuclear factor-kappa B (NF-kappaB), cyclooxygenase-2 (Cox-2), extracellular signal-regulated protein kinase (ERK1/2), mitogen-activated protein kinase (MAPK), 7,12-dimethylbenz(a)anthracene (DMBA)-initiated and 12-O-tetradecanoylphorbol-13-acetate (TPA), metalloproteinases MMP

Co-first author Yaotang Wang obtained access to the MIMIC-IV database after completing the required training on the National Institutes of Health (NIH) platform, including the courses Study data or Specimens only and Conflict of interest (certification numbers: 59979404 and 59979406)

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