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192 YoshimotoN.InoueE.SaitoK.YamayaT.TakahashiH

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Complement activation after brain ischemia is primarily mediated by circulating natural antibodies (IgM) which are binding to neo-antigens expressed on the surface of hypoxia-stressed endothelial cells [42]

The latter ones were also identified in this study as upregulated, among them Toll-like receptors (TLR2 and TLR8, IKBKB, PIK3R1, IRF5), proteins involved in NOD-like and RIG-I-like (DDX3X, ISG15, TRIM25, TMEM173, IL18, MAVS, OAS3, PKN1, NAIP) and MAPK signaling pathways (MAPK1, MAPK10, MAP2K1, CASP, ARRB, MAX, JNK, MEK1, ERK, RSK2, IKK) as well as scavenger receptors (CD68, CD44) (Supplementary Table S3)

, Myalgic encephalomyelitis/chronic fatigue syndrome is common in post-acute sequelae of SARS-CoV-2 infection (PASC): Results from a post-COVID-19 multidisciplinary clinic