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Our results thus indicated that AITC treatment led to glutathione depletion which contributed to the higher susceptibility of glutathione mutants, but the molecular mechanism behind this physiological response is likely more complicated

It is recognized as the key to destroying fatty acid hydroperoxides ( Ferrozois, a new form of regulation of cell death, is primarily attributed to cellular lipid peroxidation (LPO) due to insufficient or absent GPX4 activity, ultimately causing cell death ( 2 O 2 ) to water (Ursini and Maiorino, 2020)