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Description

aeruginosa secretes TesG via the TesABC Type I secretion system, which impairs alveolar macrophage activity by inhibiting RhoA-mediated immune signaling, thereby promoting biofilm formation and infection chronicity (Fig

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Protein synthesis and breakdown in skin and muscle: a leg model of amino acid kinetics

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Promote brighter and more even-toned skin

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VEGFR2-Fc interacted with VEGF, but not with FGF1 or FGF2 (Supplementary Fig

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For example, the members of Pseudomonas frequently isolated by H

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Tim-3/galectin-9 regulate the homeostasis of hepatic NKT cells in a murine model of nonalcoholic fatty liver disease

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