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Ribeiro AR, Nunes OC, Pereira MF, Silva AM

When local L-lactate concentrations persistently exceed physiological thresholds or when D-lactate levels abnormally rise due to methylglyoxal accumulation, and this elevated state persists beyond the acute stress window (Magistretti and Allaman, 2018), excessive histone lactylation modifications evolve from physiological transcriptional fine-tuning into irreversible pathological remodeling

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The findings indicated a significant reduction in the proportion of deaths attributable to cardiac causes (195)

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