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Writers such as histone acetyltransferases (HATs), including GCN5 and HAT family members [34], and histone methyltransferases (HMTs) like the SDG and SUVH families [35], deposit activating or repressive marks on histones [36]

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Regulation of XIAP turnover reveals a role for USP11 in promotion of tumorigenesis

In addition, except for anti-malaria drugs, menin-mixed-lineage leukemia (MLL) inhibitors used to treat MLL-rearranged leukemia were also reported able to exert an excellent cytotoxic effect on EOC cells via inducing ferroptosis [44]