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Considering that CBS deficiency is an autosomal recessive disorder and that no other alteration was detected in coding regions of the CBS gene in homocystinuric patients of this family, it is highly possible that the two novel mutations of CBS identified here are responsible for the pathogenesis of homocystinuria in this pedigree

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FIGURE 1 Dopamine Metabolism Selective degeneration of the DA neurons of the SNpc suggests that DA itself may be a source of oxidative stress (Segura-Aguilar et al., 2014)

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Mental Fatigue: NAC and glutathione protect the brain's mitochondria from oxidative damage, ensuring that neurons have the continuous ATP energy required for complex cognitive tasks