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Treatment of breast cancer cell lines with 28-homocastasterone (28-homoCS) and 24-epibrassinolide (24-epiBL) has been associated with reduced expression of cyclins involved in the G1 phase of the cell cycle

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This immunological shift was mechanistically associated with inhibition of NF-B signaling and activation of PPAR, pathways well recognized for their opposing roles in inflammation and metabolic regulation ( 2 D 3 demonstrated modest reductions in hepatic mRNA expression of the pro-inflammatory cytokines TNF- and IL-6

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