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Comprehensive metabolic profiling of inflammation indicated key roles of glycerophospholipid and arginine metabolism in coronary artery disease

3G-I), pretreatment with hemoglobin (H 2 S scavenger) was able to nullify the inhibitory impact of GYY4137 on LPS/ATP-induced proapoptosis (Supplemental Fig

These chemicals are generated from injured or stressed cells in response to ROS formation and operate as signaling factors to recruit and activate immune cells, sustaining inflammation (Albano et al., 2022)

Later, this question was further explored by Mutvei et al

This can result in the depolarization of sensory and motor nerves and the activation of mast cells