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Qazi SS, Osoria Perez A, Sam M, Leslie EM

Hypotheses 48 (5), 381386

[58] have shown that Nrf2 deficiency leads to oxidative stress and DNA lesions, accompanied by impairment of cell-cycle progression, mainly G(2)/M-phase arrest

We identified interactions with classical complement proteins and mapped binding regions in the E

HIV protease inhibitorsritonavir, indinavir, saquinavir, nelfinavir, and zidovudinewere described as ABCC1 substrate [35], suggesting that the overexpression of ABCC1 in infected cells makes these cells more resistant to chemotherapy

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