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Potentiation of antibiofilm activity of amphotericin B by superoxide dismutase inhibition

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These data suggest that the majority of LRRK2-mediated phosphorylation signal minimally resides within residues 252-359 of ArfGAP1 (GAP-F5 fragment), although additional less abundant sites are likely to exist outside of this region as suggested by higher phosphorylation of the GAP-F1 fragment (137-415