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Studies in humans and animals with non-alcoholic fatty liver disease show that CD44 plays a key role in the inflammation and liver damage in NAFLD.[ref] Other genetic studies have looked at genes in pathways known to be involved in acetaminophen metabolism, including genes related to sulfation, glucuronidation, and glutathione production

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When DAO levels are insufficient, dietary histamine accumulates in the bloodstream, leading to a state of histamine intolerance that exacerbates systemic inflammation